Question
[19]Thus, in the article on Painting, in 17 Encyc. Brit., 14th Ed. 1932, 36-64D, there is a front view nude female (Plate VIII, 2) and
[19]Thus, in the article on Painting, in 17 Encyc. Brit., 14th Ed. 1932, 36-64D, there is a front view nude female (Plate VIII, 2) and a front view of nude males and females together (Plate XXIV, 7). In the article on Sculpture in 20 Encyc. Brit. (14th Ed. 1932) 198-217, there are front views of nude females (Plate V, 4 and 6; Plate VI, 8; Plate XVIII, 2 and 4). There are also front views of nude males (Plate IV, 8; Plate XIX, 8), and of nude males and females in physical contact (Plate IV, 2; Plate V, 8; Plate VI, 2; Plate XVIII, 3). In the article on Sculpture Technique, in 20 Encyc. Brit. (14th Ed. 1932) 217-231, there are front views of nude females (Plate VII, 1; Plate IX, 4); front views of nude males (Plate I, 1; Plate II, 6 and 8; Plate V, 7; Plate IX, 2); and a nude male and nude female in physical contact (Plate VIII, 3). These pictures in the Encyclopaedia Britannica are all larger than are the pictures which appear in Parmelee's book, and several of them contra Parmelee frankly emphasize sexual subjects.
Question 1
A 25-year-old woman, pregnant in her second trimester, starts to
experience chorea and bilateral ankle arthralgia but has no past history of
rheumatic chorea. In the first hour, her erythrocyte sedimentation rate is
70. Could this be no more than chorea gravidarum?
Question 2
Is valproate as equally effective as haloperidol in the treatment of chorea,
in particular rheumatic chorea?
Question 3
Does a lesion of Guillain-Mollaret's triangle in the brain stem cause a
type of myoclonus other than symptomatic palatal myoclonus?
Question 4
1. In West's syndrome, after the fits have been suppressed, for how
long should treatment with adrenocorticotrophic hormone (ACTH)
continue?
2. Does complete suppression of resistant infantile myoclonic jerks by
ACTH characterize West's syndrome?
Question 5
Are anticholinergics the first line of treatment for primary torsion
dystonia?130f21
Question 6
Can multiple sclerosis (MS) be associated with lack of vitamin D,
lack of sunlight or low fish/cod-liver oil in the diet? By looking at the
epidemiology (none at the equator; more outside 40 latitude, both north
and south; less on top of Swiss mountains than in the Swiss valleys; more
in fishing coastal towns and in Eskimos) this seems to be very important.
Vitamin D modulates the immune system and active vitamin D given
to rats with experimental MS (acute encephalomyelitis) lowers the
monocyte count in cerebrospinal fluid (CSF) by 90% in 72 hours with
return of power to their limbs. Japanese MS patients who ate plenty of
fish were found to have vitamin-D-receptor pleomorphism. The staple
grains and cereals (wheat, barley, oats) eaten in Scandinavian and
northern European countries contain phytic acid, which blocks vitamin D
absorption, and rice is the only cereal free of phytic acid.
Are there any studies where low vitamin D levels in blood are
associated with MS relapse?
Question 7
What are the diagnostic criteria of 'definitive' multiple sclerosis (MS) - as
taught to a medical student? We have found different information from
different sources.
Question 8
How reliable is a CT-brain scan with contrast in showing MS lesions as
enhancing lesions in the presence of a contraindication to use MRI?
Question 9
Is magnetic resonance (MR) spectroscopy of value in differentiating
multiple sclerosis from cerebral autosomal dominant arteriopathy with
subcortical infarctions (CADASIL)?
Question 10
Does hemiplegia due to multiple sclerosis present with hemiparesis
rather than dense hemiplegia (which is more characteristic of a stroke)?
Other than age, what are the clinical signs that would help differentiate
between the two?
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