Defendant's driver delivered several cases of Coca Cola to the restaurant, placing them on the floor, one on top of the other, under and behind the counter, where they remained at least thirty-six hours. Immediately before the accident, plaintiff picked up the top case and set it upon a near-by ice cream cabinet in front of and about three feet from the refrigerator. She then proceeded to take the bottles from the case with her right hand, one at a time, and put them into the refrigerator. Plaintiff testified that after she had placed three bottles in the refrigerator and had moved the fourth bottle about eighteen inches from the case "it exploded in my hand." The bottle broke into two jagged pieces and inflicted a deep five-inch cut, severing blood vessels, nerves and muscles of the thumb and palm of the hand. Plaintiff further testified that when the bottle exploded, "It made a sound similar to an electric light bulb that would have dropped. It made a loud pop." Plaintiff's employer testified, "I was about twenty feet from where it actually happened and I heard the explosion." A fellow employee, on the opposite side of the counter, testified that plaintiff "had the bottle, I should judge, waist high, and I know that it didn't bang either the case or the door or another bottle ... when it popped. It sounded just like a fruit jar would blow up

Question 1

I want to ask something about cerebrovascular accident (CVA). Can you

please tell me how we can rapidly pinpoint the exact anatomical site of the

neurological deficit using physical findings in the emergency room?

Question 2

Why do you treat dissection of the carotid artery with an anticoagulant

in the acute management of stroke secondary to dissection? To me this

seems paradoxical as it would increase the severity of dissection.

Question 3

Last week, in a neurology viva, I was asked about the indications for

heparinization in patients with a stroke. I want to know when I can stop

heparin and what test I should use for assessing its therapeutic range.

Question 4

Has heparin a role in the management of acute ischaemic stroke not

accompanied by atrial fibrillation?

Question 5

1. In the treatment of a stroke, does low-molecular-weight heparin

(LMWH) have an advantage over heparin?

2. In an ischaemic stroke in evolution, for how long should heparin be

administered?

Question 6

Can streptokinase be used in acute cerebral infarction and, if so, what is

the dose?

Question 7

There seems now to be a consensus about starting aspirin therapy in

acute ischaemic strokes as early as possible. Why has this changed from

Question 8

I understand that a loading dose of clopidogrel 600-900 mg can be given

to ischaemic stroke in evolution and can stop the evolving deficit. Would

you agree?

Question 9

Is there any rationale for giving patients with recurrent strokes a

combination of aspirin and anticoagulant?

Question 10617021

1. Does a dipyridamol-aspirin combination have any superiority over

aspirin alone in the secondary prevention of a stroke?

2. Is an aspirin plus anticoagulant combination superior to a dipyridamol

aspirin combination in the treatment of recurrent ischaemic stroke not

controlled by aspirin alone?