FOR STATISTICS EXPERTS

define the central tenancy in atatistics

First, for example, inBrown v. Municipality of Anchorage,^[15]the Supreme Court of Alaska struck down a prostitution loitering ordinance in part because a formerly convicted prostitute could be convicted again simply based on (1) the act of loitering, and (2) his or her status as a known prostitute, without committing any other overt act demonstrating that he or she had induced, enticed, solicited, or procured another to commit prostitution.^[16]The court concluded that, even though it suggests the purposes to engage in prostitution, this additional circumstance did not qualify as an adequate guideline the ordinance's vagueness left too much discretion in the hands of the police, who could apply it arbitrarily.^[17]Because CCO 12.08.030 does not have even the unconstitutionally vague "known prostitute" element of the Alaska ordinance, it leaves authorities with an even greater amount of discretion.

Question 1

I want to ask something about cerebrovascular accident (CVA). Can you

please tell me how we can rapidly pinpoint the exact anatomical site of the

neurological deficit using physical findings in the emergency room?

Question 2

Why do you treat dissection of the carotid artery with an anticoagulant

in the acute management of stroke secondary to dissection? To me this

seems paradoxical as it would increase the severity of dissection.

Question 3

Last week, in a neurology viva, I was asked about the indications for

heparinization in patients with a stroke. I want to know when I can stop

heparin and what test I should use for assessing its therapeutic range.

Question 4

Has heparin a role in the management of acute ischaemic stroke not

accompanied by atrial fibrillation?

Question 5

1. In the treatment of a stroke, does low-molecular-weight heparin

(LMWH) have an advantage over heparin?

2. In an ischaemic stroke in evolution, for how long should heparin be

administered?

Question 6

Can streptokinase be used in acute cerebral infarction and, if so, what is

the dose?

Question 7

There seems now to be a consensus about starting aspirin therapy in

acute ischaemic strokes as early as possible. Why has this changed from

Question 8

I understand that a loading dose of clopidogrel 600-900 mg can be given

to ischaemic stroke in evolution and can stop the evolving deficit. Would

you agree?

Question 9

Is there any rationale for giving patients with recurrent strokes a

combination of aspirin and anticoagulant?

Question 10

1. Does a dipyridamol-aspirin combination have any superiority over

part 2

Question 1

What is the best treatment for rubral tremors besides treating the aetiology?

Question 2

What is meant by 'inversion of reflexes'? I have found this term in a few membership exams.

Question 3

Is it possible for patients with posterior column lesions to be suffering from allodynia, with pain on pressure to different musculoskeletal points? Or is this more likely to be caused by fibromyalgia?

Question 4

How do the clinical success rates of gabapentin and carbamazepine compare?

Question 5

What clinical tests can be done to determine dissociative sensory loss?

Question 6

What is the recommended dose of urograffin before performing contrastenhanced computed tomography? How far in advance should this be administered before imaging when an intracerebral abscess or glial tumour is suspected?

Question 7

How many urografin ampoules (76% concentration) should be administered before a CT brain scan with contrast searching for a mass lesion, and how many minutes before imaging should these be injected?

Question 8

1. What is the difference between fluid-attenuated inversion recovery (FLAIR) and T2-weighted MRI scans?

2. What is the advantage of magnetization transfer pulse over a FLAIR MRI scan?

Question 9

Does lumbosacral MRI refer to lumbosacral spines or to lumbosacral cord segments?

Question 10

What type of painful stimuli can be applied in calculating the Glasgow Coma Scale for both motor and eye-opening responses, and how should these be applied?

Question 1

What is the best treatment for rubral tremors besides treating the aetiology?

Question 2

What is meant by 'inversion of reflexes'? I have found this term in a few membership exams.

Question 3

Is it possible for patients with posterior column lesions to be suffering from allodynia, with pain on pressure to different musculoskeletal points? Or is this more likely to be caused by fibromyalgia?

Question 4

How do the clinical success rates of gabapentin and carbamazepine compare?

Question 5

What clinical tests can be done to determine dissociative sensory loss?

Question 6

What is the recommended dose of urograffin before performing contrastenhanced computed tomography? How far in advance should this be

administered before imaging when an intracerebral abscess or glial

tumour is suspected?

Question 7

How many urografin ampoules (76% concentration) should be

administered before a CT brain scan with contrast searching for a mass

lesion, and how many minutes before imaging should these be injected?

Question 8

1. What is the difference between fluid-attenuated inversion recovery

(FLAIR) and T2-weighted MRI scans?

2. What is the advantage of magnetization transfer pulse over a FLAIR

MRI scan?

Question 9

Does lumbosacral MRI refer to lumbosacral spines or to lumbosacral cord

segments?

Question 10

What type of painful stimuli can be applied in calculating the Glasgow

Coma Scale for both motor and eye-opening responses, and how should

these be applied?Question 1

What is the best treatment for rubral tremors besides treating the aetiology?

Question 2

What is meant by 'inversion of reflexes'? I have found this term in a few membership exams.

Question 3

Is it possible for patients with posterior column lesions to be suffering from allodynia, with pain on pressure to different musculoskeletal points? Or is this more likely to be caused by fibromyalgia?

Question 4

How do the clinical success rates of gabapentin and carbamazepine compare?

Question 5

What clinical tests can be done to determine dissociative sensory loss?

Question 6

What is the recommended dose of urograffin before performing contrastenhanced computed tomography? How far in advance should this be administered before imaging when an intracerebral abscess or glial tumour is suspected?

Question 7

How many urografin ampoules (76% concentration) should be administered before a CT brain scan with contrast searching for a mass lesion, and how many minutes before imaging should these be injected?

Question 8

1. What is the difference between fluid-attenuated inversion recovery (FLAIR) and T2-weighted MRI scans?

2. What is the advantage of magnetization transfer pulse over a FLAIR MRI scan?

Question 9

Does lumbosacral MRI refer to lumbosacral spines or to lumbosacral cord segments?

Question 10

What type of painful stimuli can be applied in calculating the Glasgow Coma Scale for both motor and eye-opening responses, and how should these be applied?

Question 1

What is the best treatment for rubral tremors besides treating the aetiology?

Question 2

What is meant by 'inversion of reflexes'? I have found this term in a few membership exams.

Question 3

Is it possible for patients with posterior column lesions to be suffering from allodynia, with pain on pressure to different musculoskeletal points? Or is this more likely to be caused by fibromyalgia?

Question 4

How do the clinical success rates of gabapentin and carbamazepine compare?

Question 5

What clinical tests can be done to determine dissociative sensory loss?

Question 6

What is the recommended dose of urograffin before performing contrastenhanced computed tomography? How far in advance should this be

administered before imaging when an intracerebral abscess or glial

tumour is suspected?

Question 7

How many urografin ampoules (76% concentration) should be

administered before a CT brain scan with contrast searching for a mass

lesion, and how many minutes before imaging should these be injected?

Question 8

1. What is the difference between fluid-attenuated inversion recovery

(FLAIR) and T2-weighted MRI scans?

2. What is the advantage of magnetization transfer pulse over a FLAIR

MRI scan?

Question 9

Does lumbosacral MRI refer to lumbosacral spines or to lumbosacral cord

segments?

Question 10

What type of painful stimuli can be applied in calculating the Glasgow

Coma Scale for both motor and eye-opening responses, and how should

these be applied?