Question 49 Several recent studies comparing aspirin to warfarin have been published.If a patient has had a first myocardial infarction (MI), would you prescribe warfarin as a secondary prevention of coronary heart disease? Question 50: Is it true that the ApoA will in the future:Will the ApoB ratio be used more effectively than low-density lipoprotein (LDL) to predict the risk of coronary artery disease? What exactly is the connection between C-reactive protein (CRP) and coronary artery disease, as asked in Question 51? Question 52: In angina pectoris, why does the pain in the chest spread to the left side, including the left arm and back? In spite of the fact that both non-ST-segment elevation myocardial infarctions (nSTE-MI) and ST-elevated myocardial infarctions (STE-MI) are caused by a thrombus, for which thrombolytic therapy is highly recommended, could you please explain why thrombolytic therapy is not recommended in cases of unstable angina?Is it possible that intracranial hemorrhage is more common in nSTE-MI patients with unstable angina than in ST-segment elevation MI patients? Question 54: Stable angina, nonST-segment elevation myocardial infarctions, and ST-elevated myocardial infarctions (STEMI) are all included in the term "acute coronary syndrome." Please provide an explanation of the uses of clopidogrel for acute coronary syndrome (ACS).If aspirin is contraindicated, should it be taken alone or with it?Are there any studies that combine both with either unfractionated or low-molecular-weight heparin (LMWH)?How much time should clopidogrel be taken? Question 56: In the treatment of acute inferior wall myocardial infarction, what role do intravenous fluids play? Question 57: In patients with post-MI angina and ischaemic stroke, is it beneficial to take aspirin in addition to clopidogrel?Would you recommend that we stop using this combination in our hospital unit as a result of the MATCH trial's finding that it was not beneficial? Question 58: Would highly elevated cardiac enzymes indicate thrombolysis in a patient with typical myocardial infarction (MI) chest pain and no other thrombolysis criteria? Question 59 From Conn's Current Therapy and Swanson's Family Practice, I am unable to determine the role of an acetylcholinesterase inhibitor in post-myocardial infarction.Can you assist?