The diathesis stress approach to mental illness proposes that neither environmental stress alone nor genetic factors alone are enough to produce mental illness. Instead, both environmental stress and genetic predisposition to mental illness are required for mental illnesses like schizophrenia and depression to be expressed. In a recent test of this idea (Sachs, Ni, & Caron, 2015), either normal rats or rats that were genetically modified to have low levels of the neurotransmitter serotonin received either no social stress or a social stress treatment. Researchers measured the number of social interactions produced by subjects after the stress treatment. Data like those observed by the author are listed below. Factor A: Stress Factor B: Gene Low serotonin High serotonin 0 15 8 17 Social Stress 11 23 5 14 8 16 10 23 19 10 12 16 None 22 17 20 17 15 23 20 19 Use a twofactor ANOVA with a = .05 to evaluate the significance of the main effects and the interaction. Compute the following. Factor A: Stress Factor B: Gene Social Stress None Fill in the missing values of the following ANOVA table. Note: Do not round the MS values, but round the F statistics to three decimal places. Low serotonin M = 7 T = 42 SS = 374 M = 18 T = 108 55 = 2,014 2X2 = 6,236 High serotonin M = 18 T = 108 SS = 2,024 M = 17 T = 102 SS = 1,824 Source MS F Between treatmen Factor A (stress) ~ - 150 9375 Factor B (gene) 150 150 9.375 A x B Interaction 216 216 13.500 Within treatments 320 20 16 Total 836 23