handle this please

Question 1

Can pyramidal tract lesions present as false localizing signs in benign

intracranial hypertension?

Question 2

How often can increased intracranial hypertension be present without

papilloedema on examination?

Question 3

Which is the most effective method of reducing raised intracranial

pressure: mannitol, steroids or ventilation?

Question 4

In the case of pseudotumour cerebri, if this is proved to be due to sagittal

sinus thrombosis with no explanation for the thrombosis, should the

patient receive lifelong anticoagulation treatment?

Question 5

Does digitalis help reduce cerebrospinal fluid (CSF) formation,

specifically in the treatment of resistant benign intracranial hypertension?

Question 6

Is it safe to combine hydrochlorothiazide, amiloride, acetazolamide

and digitalis (125 g per day) in a hypertensive patient with idiopathic

intracranial hypertension (pseudotumour cerebri)?

Question 7

What is the best indicator for monitoring the efficacy of treatment or the

progress of the disease in pseudotumor cerebri? Is it by the disappearance

of papilloedema, the relief of headache or by frequent visual field

examination or cerebrospinal fluid (CSF) pressure?

Question 8

What is the definition of 'bursting' when describing a headache?

Question 9

1. What drugs, other than tricyclic antidepressants, can be used in

prophylaxis for daily tension headache?

2. Is propranolol an effective treatment?

Question 10

Are maprotiline and imipramine as effective as amitriptyline in the

treatment of tension headache?